Addictive behavior
Addiction and dependence glossary[1][2][3] | |
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An addictive behavior is a behavior, or a stimulus related to a behavior (e.g., sex or food), that is both rewarding and reinforcing, and is associated with the development of an addiction. There are two main forms of addiction: substance use disorders (including alcohol, tobacco, drugs and cannabis) and behavioral addiction.[4] Any activity that activates the reward system in your brain has the potential to cause behavioral addictions (including sex, gambling, eating and shoplifting). The parallels and distinctions between behavioral addictions and other compulsive behavior disorders like bulimia nervosa and obsessive-compulsive disorder (OCD) are still being researched by behavioral scientists.[5]
The Addiction is classified as a chronic brain disorder by the American Society of Addiction Medicine (ASAM).[5] There is a medical model which perceives addictive behavior as a disease that is caused by uncontrollable and recessive drug use overtimes and the addict barely has control of it.[6] There are several reasons why people develop an addiction. A predisposition to the addictive qualities of substances may be inherited by some people, making it a genetic circumstance. Another cause for addictions could be the environment. Whether or not someone develops substance use problems can be influenced by their home and neighborhood, as well as the attitudes of their peers, family, and culture on substance use. Another cause of developing an addiction could be related to mental health issues: over 50% of individuals with substance use disorders have experienced mental health issues at some point in their lives. Even moderate substance usage might exacerbate mental health issues in individuals.The other view is from the moral standpoint which regards addictive behavior as an intentional choice was freely made by the addict.[6]
Compulsion vs addiction
Compulsions are the basis of addictions. Reward is one major distinction between compulsion in addicts and compulsion as it is experienced in obsessive-compulsive disorder. An addiction is, by definition, a form of compulsion, and involves operant reinforcement. For example, dopamine is released in the brain's reward system and is a motive for behaviour (i.e. the compulsions in addiction development through positive reinforcement).[7]
There are two main differences between compulsion and addiction. Compulsion is the need and desire to do something or carry out a task repetitively or persistently. Whereas addiction is defined by the following step after compulsion where an individual takes action on a compulsion to feel pleasure and satisfaction (the action is known as compulsive behavior). Notably, for addicts, compulsive behavior can lead to “persistent changes in the brain structures and functions” which creates a cycle of motivation for their behavior that is absent in OCD.[8]
Compulsions (and compulsive behavior) do not necessarily include pleasure for the individual but addictive behavior does. In contrast, someone who experiences a compulsion as part of obsessive-compulsive disorder may not perceive anything rewarding from acting on the compulsion. Often, it is a way of dealing with the obsessive part of the disorder, resulting in a feeling of relief (i.e., compulsions may also arise through negative reinforcement).[9]
Correlation between different personality traits and addictive behaviors online
The study indicates that there is a difference in personality traits for addictive behaviors that is associated with distinctive online tasks.[10] Namely, higher neuroticism and less conscientiousness have a correlation with internet addiction overall;[10] less conscientiousness and low openness to experience are connected with addiction to video games,[10] neuroticism and extraversion were linked with social networking addiction.[10]
Development of addictive behaviors in adolescents
Drug or alcohol-induced addictive behaviors in adolescents have been linked to models discussing a stronger sensitization of the appetitive response and disrupted inhibitory control.[11] In early adolescence, individuals lack the motivation to control appetitive motivational tendencies.[11] The presence of positive and negative reinforcers seem to increase alcohol consumption, portraying a positive correlation between motives and alcohol consumption.[11] After initiating drinking, there seems to be an increase in appetitive motivation to drink and a negative effect on controlled regulatory processes.[11] The imbalance between these factors lead to increased substance use.[11]
Therapy for addictions
Addictions might give an individual a sense of power, confidence, endorsement, or other feelings that they might not attain in their real life.[12] Psychotherapy is a way to help people distinguish rough feelings and distress factors to adapt and grow.[12] People that have trouble with addictive behaviors are unlikely to regulate those actions by themselves unless someone leads them to work on addressing the root of their addictive behaviors, such as trauma, stress, anxiety, and so on.[12] There are a lot of types of therapy that can help people address the addictive behaviors which they want to alter.[12] An individual who has an addictive behavior would most likely have the biggest advantage from consulting with a therapist that is recognized by the medical institution or organization to be the expert in the particular area of addiction or compulsion in terms of potential causes.[12]
See also
- Addiction
- ANNK1 and addictive behaviors
- Addiction vulnerability
- Behavioral addiction
- Habituation
- Substance dependence
References
- ^ Nestler EJ (December 2013). "Cellular basis of memory for addiction". Dialogues in Clinical Neuroscience. 15 (4): 431–443. PMC 3898681. PMID 24459410.
Despite the importance of numerous psychosocial factors, at its core, drug addiction involves a biological process: the ability of repeated exposure to a drug of abuse to induce changes in a vulnerable brain that drive the compulsive seeking and taking of drugs, and loss of control over drug use, that define a state of addiction. ... A large body of literature has demonstrated that such ΔFosB induction in D1-type [nucleus accumbens] neurons increases an animal's sensitivity to drug as well as natural rewards and promotes drug self-administration, presumably through a process of positive reinforcement ... Another ΔFosB target is cFos: as ΔFosB accumulates with repeated drug exposure it represses c-Fos and contributes to the molecular switch whereby ΔFosB is selectively induced in the chronic drug-treated state.41 ... Moreover, there is increasing evidence that, despite a range of genetic risks for addiction across the population, exposure to sufficiently high doses of a drug for long periods of time can transform someone who has relatively lower genetic loading into an addict.
- ^ Malenka RC, Nestler EJ, Hyman SE (2009). "Chapter 15: Reinforcement and Addictive Disorders". In Sydor A, Brown RY (eds.). Molecular Neuropharmacology: A Foundation for Clinical Neuroscience (2nd ed.). New York: McGraw-Hill Medical. pp. 364–375. ISBN 978-0-07-148127-4.
- ^ Volkow ND, Koob GF, McLellan AT (January 2016). "Neurobiologic Advances from the Brain Disease Model of Addiction". New England Journal of Medicine. 374 (4): 363–371. doi:10.1056/NEJMra1511480. PMC 6135257. PMID 26816013.
Substance-use disorder: A diagnostic term in the fifth edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM-5) referring to recurrent use of alcohol or other drugs that causes clinically and functionally significant impairment, such as health problems, disability, and failure to meet major responsibilities at work, school, or home. Depending on the level of severity, this disorder is classified as mild, moderate, or severe.
Addiction: A term used to indicate the most severe, chronic stage of substance-use disorder, in which there is a substantial loss of self-control, as indicated by compulsive drug taking despite the desire to stop taking the drug. In the DSM-5, the term addiction is synonymous with the classification of severe substance-use disorder. - ^ "Top 10 Most Common Addictions in the U.S." Addiction Center. Retrieved 2021-03-07.
- ^ a b "Addiction". Cleveland Clinic. Retrieved 2023-11-07.
- ^ a b Henden, Edmund; Melberg, Hans Olav; Røgeberg, Ole Jørgen (2013). "Addiction: Choice or Compulsion?". Frontiers in Psychiatry. 4: 77. doi:10.3389/fpsyt.2013.00077. ISSN 1664-0640. PMC 3736117. PMID 23966955.
- ^ Solinas, Marcello (January 30, 2020). "Dopamine and addiction: What have we learned from 40 years of research". Journal of Neural Transmission. 126 (4).
- ^ Henden, Edmund; Melberg, Hans Olav; Røgeberg, Ole Jørgen (2013-08-07). "Addiction: Choice or Compulsion?". Frontiers in Psychiatry. 4: 77. doi:10.3389/fpsyt.2013.00077. ISSN 1664-0640. PMC 3736117. PMID 23966955.
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: CS1 maint: unflagged free DOI (link) - ^ "NIMH » Obsessive-Compulsive Disorder: When Unwanted Thoughts or Repetitive Behaviors Take Over". www.nimh.nih.gov. Retrieved 2020-10-13.
- ^ a b c d Wang, Chong-Wen; Ho, Rainbow T.H.; Chan, Cecilia L.W.; Tse, Samson (March 2015). "Exploring personality characteristics of Chinese adolescents with internet-related addictive behaviors: Trait differences for gaming addiction and social networking addiction". Addictive Behaviors. 42: 32–35. doi:10.1016/j.addbeh.2014.10.039. ISSN 0306-4603. PMID 25462651.
- ^ a b c d e Wiers, Reinout W.; Bartholow, Bruce D.; van den Wildenberg, Esther; Thush, Carolien; Engels, Rutger C.M.E.; Sher, Kenneth J.; Grenard, Jerry; Ames, Susan L.; Stacy, Alan W. (February 2007). "Automatic and controlled processes and the development of addictive behaviors in adolescents: A review and a model". Pharmacology Biochemistry and Behavior. 86 (2): 263–283. doi:10.1016/j.pbb.2006.09.021. ISSN 0091-3057. PMID 17116324. S2CID 18979706.
- ^ a b c d e Chabert, Catherine (2015-03-04), "Obsessions, addictions, compulsions : contraintes narcissiques et fantasme d'inceste", Les travaux forcés de la répétition, Petite bibliothèque de psychanalyse, Presses Universitaires de France, pp. 19–40, doi:10.3917/puf.andre.2015.03.0019, ISBN 9782130653455, retrieved 2022-04-22